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Pre-index HbA1c and BMI were also balanced between the cohorts post-matching (i.e SMD of -0.021 and 0.055, respectively), suggesting that the PSM process had effectively identified control patients who had similar profiles as GLP-1 patients at index date for the study
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Therefore, further investigation to identify which epigenetic factors, TFs or inflammatory mediators are directly responsible for establishing and maintaining these specific epigenetic memories in vivo would provide deeper mechanistic insights and shed light on potential therapeutic targets to prevent rUTIs and/or reverse the epigenetic imprinting that leads to increased susceptibility to recurrent disease
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