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5.1.4 Gene therapy and genome editing Recent advances in gene therapy and genome editing offer promising approaches to modulate ferroptosis in OA by targeting critical regulators such as GPX4, SLC7A11, and epigenetic modifiers

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How SLM+ Complements This Framework While GLP-1 medications dominate the pharmaceutical space, theres growing recognition of adjunctive strategies that can support hormone signaling and metabolic efficiencyespecially in patients who: Cant access or tolerate dual/triple agonists Are at risk of plateauing Need long-term metabolic support after discontinuing GLP-1s SLM+ was developed to fill this gap

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thus, mitochondrial dysfunction may also be involved (Kerckhove et al., 2012) have reported in rat sciatic nerve how bortemozib, as other antitumor drugs, causes significant deficits in complex I and II of ETC, as well as in ATP production, at two time points (pain onset -day 7- and peak pain -day 35-)