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The data are preclinical, human evidence is absent, and its pharmacology while mechanistically interesting has not been validated in clinical settings

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Sci Rep 11 , 20765 (2021)

Ge JB, Fan WH, Zhou JM, Shi HM, Ji FS, Wu Y, et al

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Indeed, a number of mitochondrial abnormalities are seen in both disease-affected individuals and animal models, including reduced calcium buffering capability, compromised bioenergetics, increased oxidative stress, and aberrant trafficking and dynamics [47]