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In toxin-based models (MPTP or 6-OHDA), flavonoids prevent dopaminergic neuron dysfunction by preserving dopamine levels, reducing oxidative damage, and maintaining tyrosine hydroxylase (TH) protein expression (18)

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In this review, we discuss how these two G-protein coupled receptor (GPCR) ligands may improve L-DOPA-based treatment with minimized side effects or provide alternative nondopaminergic options for patients who would benefit from early treatment