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Diabetologia, 38 (12), 14251433

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This is not surprising because the cellular and molecular adaptations underlying tolerance almost certainly do not have a unitary mechanism, and different cell lines, different neurons and neuronal compartments (i.e., terminal versus somatic compartments) may express different levels of regulatory proteins

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Cell growth, controlled by sodium-hydrogen ion exchangers (NHEs), induces MIC-dependent activation of the PI3K/Akt pathway, which in turn supports the cytoplasmic localization of the cell cycle inhibitor p27 Kip1 , resulting in tumor cell proliferation [142]

Rahlfs, S., Fischer, M