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Based on the important role of ferroptosis in AD pathogenesis, inhibiting ferroptosis has become a significant strategy to reduce neuronal damage in AD in preclinical studies
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MitoQ, a mitochondria-penetrating ubiquinone derivative, restores mitochondrial redox balance and significantly alleviates cachexia-induced muscle wasting in C26 tumor-bearing mice without affecting tumor burden
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Regulation of T-cell function by endogenously produced angiotensin II