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Recently, it was demonstrated that ATP-P2X7-inflammasome-caspase 1-IL-1/18 axis played a role in smoke-induced airway inflammation in healthy smokers or COPD patients as well as murine COPD models, with increased activation of P2X7 receptor/caspase 1 and elevated IL-1 and IL-18 levels (56)
Funding This work was supported in part by the National Institute on Drug Abuse of the National Institutes of Health and the Center for Tobacco Products of the US Food and Drug Administration (under Award Number P50-DA-036107)
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When it enters the human body, it stimulates the nervous system, causing an increase in pulse rate and blood pressure
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