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Rutledge SM, Asgharpour A

A growing number of studies examining how dysbiosis might drive NAFLD have identified a number of plausible mechanisms, including changes in short-chain fatty acids (SCFAs) metabolism, increased intestinal permeability and lipopolysaccharides (LPS) activation of toll-like receptors (TLRs) and inflammasomes, endogenous ethanol production, decreased choline availability, and trimethylamine production (69)

Associations between liver histology and severity of the metabolic syndrome in subjects with nonalcoholic fatty liver disease

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It is important to note that the complex interplay between smoking, viral hepatitis, and other cofactors like alcohol necessitates the further research