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It may be the TMAO, the trimethylamine oxide

Inhibition of NNMT rebalances cellular methyl economy and NAD salvage flux, with downstream effects on adipocyte energetics, lipid handling, and insulin action observed in preclinical systems [13]

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Modern pharmacological agents such as glucagon-like peptide-1 (GLP-1) receptor agonists and combination glucose-dependent insulinotropic polypeptide (GIP)/GLP-1 receptor agonists have also been shown to improve cardiovascular outcomes in patients with obesity

This is because its quickly digested

ISME J 5(1):122130