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In pregnant mice given 6, 68, 460, or 760 mcg/kg/day exenatide during fetal organogenesis, skeletal variations associated with slowed fetal growth, including changes in number of rib pairs or vertebral ossifications sites, and wavy ribs were observed at 760 mcg/kg/day, a dose that produced maternal toxicity and yielded systemic exposure 200-times the human exposure resulting from the MRHD of BYDUREON BCISE based on AUC comparison

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The results reported in Figure 1b show that the hypertension derived by administration of methylcellulose 8 causes a dramatic increase of iNOS, which is involved in the execution of the apoptotic pathway (lane 3)

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This matches the 20mg vial sweet spot (200mcg per unit) while providing 2.5 weeks of peptide at the maximum 12mg dose

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