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Based on these findings the authors concluded 'that central GLP-1 is a new physiological mediator of satiety' (Turton, O'Shea, Gunn)

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This receptor responds well to sustained levels, meaning split dosing should theoretically maintain adequate appetite suppression

In animal studies, GLP-1RAs consistently reduced drug intake, weakened dopamine release in reward neural networks, and reduced the probability of relapse

18 The increased cAMP can further act on protein kinase A (PKA) and guanine nucleotide exchange protein (EPAC), and the activated PKA/EPAC can affect the activated PKA/EPAC can affect the concentration of K + and Ca 2+ ions inside and outside the cell, 19 which in turn promotes insulin secretion

Nat Rev Endocrinol 2020