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This reduced lifespan is largely attributable to increased cardiovascular disease (Ryan 2015

Consistent with our cellular data, infection of HeLa cells with the wild-type S

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The present study was initiated to investigate whether intestinal oxidative stress occurs in patients with compensated liver cirrhosis and thereby could be an underlying mechanism contributing to the epithelial barrier dysfunction observed in our previous study,28 which potentially may increase the risk of progression towards decompensated cirrhosis

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