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These findings support the concept that the microbiota indirectly modulates -cell subpopulations through the adipose tissueinflammationinsulin resistance and SCFA/LPSincretins-cells axes, even though direct single-cell studies demonstrating differential responses of individual -cell subtypes to microbiota-derived metabolites are not yet available ( via the MyD88 or TRIF adaptors, leading to NF-B activation and induction of pro-inflammatory genes encoding IL-1, IL-6, and TNF- (Lumeng, 2013)

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